Learning biology is hard because biological systems are very complex. Given that complexity, biologists are generally happy to search keys under streetlights.
A lot of the important arguments when it comes to longevity are about making guesses about the territory that’s not under the streetlights. The knowledge we have about biology is already quite large, but it’s important to always keep in mind that it’s far of from being complete.
While learning biology your task is to both learn basic concepts to make sense of other knowledge and also learn about the system of how the knowledge gets produced.
In Core Pathways of Aging, John Wentworth for example makes claims about many people in the field approaching the subject wrong. You want to understand claims like that about the nature of the information landscape.
One aspect of the article is that he talks about transposons as potentially being a very important part of aging. DNA sequencing that can only look at short reads like Ilumnia sequencing, can’t tell you how much transposons doublicated. Given that the subject was hard to study, biologist essentially proclaimed that the existing transposons in the human genome don’t do much.
Learning biology is hard because biological systems are very complex. Given that complexity, biologists are generally happy to search keys under streetlights.
A lot of the important arguments when it comes to longevity are about making guesses about the territory that’s not under the streetlights. The knowledge we have about biology is already quite large, but it’s important to always keep in mind that it’s far of from being complete.
While learning biology your task is to both learn basic concepts to make sense of other knowledge and also learn about the system of how the knowledge gets produced.
In Core Pathways of Aging, John Wentworth for example makes claims about many people in the field approaching the subject wrong. You want to understand claims like that about the nature of the information landscape.
One aspect of the article is that he talks about transposons as potentially being a very important part of aging. DNA sequencing that can only look at short reads like Ilumnia sequencing, can’t tell you how much transposons doublicated. Given that the subject was hard to study, biologist essentially proclaimed that the existing transposons in the human genome don’t do much.