I expect that knowing you’re having anaphylaxis without a solution is already reasonably close to the upper end of psychological stress, and you can’t add that much more. The reason the epinephrine concentrations are so much higher in cardiac arrest patients is not because cardiac arrest is psychologically stressful, it’s because epinephrine release is triggered by hypoxia.
I’m not sure that it does it fast enough. You likely need high CO2 blood concentration to get your epinephrine and I’m not sure you get that in the normal anaphylaxis.
At the moment I have another open question about whether anaphylaxis shock might happen in situations where the body somehow doesn’t produce as much epinephrine as it should and therefore a feedback process in the body gets out of wrack.
I expect that knowing you’re having anaphylaxis without a solution is already reasonably close to the upper end of psychological stress, and you can’t add that much more. The reason the epinephrine concentrations are so much higher in cardiac arrest patients is not because cardiac arrest is psychologically stressful, it’s because epinephrine release is triggered by hypoxia.
This suggests that blocking the ability of the person to breath for a minute might be a way to push epinephrine concentrations.
Doesn’t anaphylaxis already do this?
I’m not sure that it does it fast enough. You likely need high CO2 blood concentration to get your epinephrine and I’m not sure you get that in the normal anaphylaxis.
At the moment I have another open question about whether anaphylaxis shock might happen in situations where the body somehow doesn’t produce as much epinephrine as it should and therefore a feedback process in the body gets out of wrack.