The metabolically privileged don’t believe in metabolic privilege, since they are able to lose weight by trying!
Some of us do believe in it since we are able to stay very thin without trying. I have never dieted and never needed to.
But, we probably don’t post very much on diet blogs.
I come from a family of thin people who eat fairly unhealthily but are quite active. When I first stopped living with my parents, I basically stopped exercising and ate even more unhealthily. I became very unfit in the sense of e.g., not being able to run a block without getting out of breath, but gained very little weight. So I figure the causation is probably not mainly exercise → thinness, but more on the lines of genes → (thinness & athleticism) or genes → thinness → athleticism.
Americans who have grown up in at least moderate financial security have developed astounding rates of obesity. People who grew up in Nazi-occupied countries who were malnourished as children also developed astoundingly high obesity rates as adults. From the evidence I’ve seen, genetics is over-emphasized as the missing factor in almost every medical theory before enough is known to know better. While income correlates with obesity, it does not explain the physiological mechanism through which poorer people (relative wealth may seem to mean much more than absolute wealth, interestingly) have a much harder time staying healthy.
It seems much more plausible that both semi-adaptable epigenomic variation and multi-generational lifestyle adaptions play bigger roles in generating familial and social trends of obesity. The nutrition, gut health, and overall health of BOTH parents contributes to the making of a child, and the mother’s health strongly affects it from then until birth, after which point colostrum and then breast milk will continue to play a direct parent-to-child role in the young one’s development.
Though there is no conclusive research that I’m aware of, it is probable that children establish certain growth limitations based on signals about nutrient availability received directly from their parents during conception and then from the mother during pregnancy and breastfeeding (variances of conveyed gut flora could be the mechanism here). Then, lifestyle and its epigenomic effects as normalized during childhood continues to play probably the same-seeming role since parents will tend to feed their children the same things they eat.
Anthropologically, going back a mere few hundred years there were no cultures anywhere in the world suffering obesity epidemics, so it doesn’t make sense to attribute variance too strongly to genetics. Historically, humans have survived healthfully on almost any combination of macronutrients while the main variant between healthy civilizations seems to have been micronutrients. Since studies generally don’t account in any fashion for idiosyncratic in-utero environment or for epigenetic variations among individuals, it could turn out that a vast amount of nutritional research is entirely worthless.
E.g. clinical studies of nutrition among populations could depend entirely on sociological factors about the last generation’s diet than about the objective value of macro-nutrients (which, in my opinion, should never be claimed as the object of a study as if removed from the context of the foods they are a part of).
The father’s health can play a role after conception as well since beneficial gut bacteria, in the least, can be transferred through saliva & sex. Additionally, since these gut bacteria build up multi-generationally, it could be that antibiotic treatment seriously impairs the functioning of newborns, especially if they don’t have probiotic sources in their diet (the best of which is breastmilk from a biotics-rich mother!).
So I figure the causation is probably not mainly exercise → thinness
Which ought to be nearly obvious to anyone who has compared the calorie expenditures of common physical activities with the calorie contents of common foodstuffs. (Yes, increasing muscle mass increases thermogenesis (but so does caffeine) and I personally feel that doing abs help me feel less hungry because they kind of compress my stomach (but so does wearing higher-rise trousers and pulling their belt tighter), but those are second-order effects.)
I personally feel that doing abs help me feel less hungry because they kind of compress my stomach (but so does wearing higher-rise trousers and pulling their belt tighter)
This is also observed when wearing back-braces and corsets over the long term. In the corset-wearing/waist-training community particularly, some people have observed that without significant changes in behavior, corsets may decrease appetite; the actual effect is of course highly variable, but it’s frequent enough to be conventional wisdom in that community, so.
Some of us do believe in it since we are able to stay very thin without trying. I have never dieted and never needed to.
But, we probably don’t post very much on diet blogs.
I come from a family of thin people who eat fairly unhealthily but are quite active. When I first stopped living with my parents, I basically stopped exercising and ate even more unhealthily. I became very unfit in the sense of e.g., not being able to run a block without getting out of breath, but gained very little weight. So I figure the causation is probably not mainly exercise → thinness, but more on the lines of genes → (thinness & athleticism) or genes → thinness → athleticism.
Americans who have grown up in at least moderate financial security have developed astounding rates of obesity. People who grew up in Nazi-occupied countries who were malnourished as children also developed astoundingly high obesity rates as adults. From the evidence I’ve seen, genetics is over-emphasized as the missing factor in almost every medical theory before enough is known to know better. While income correlates with obesity, it does not explain the physiological mechanism through which poorer people (relative wealth may seem to mean much more than absolute wealth, interestingly) have a much harder time staying healthy.
It seems much more plausible that both semi-adaptable epigenomic variation and multi-generational lifestyle adaptions play bigger roles in generating familial and social trends of obesity. The nutrition, gut health, and overall health of BOTH parents contributes to the making of a child, and the mother’s health strongly affects it from then until birth, after which point colostrum and then breast milk will continue to play a direct parent-to-child role in the young one’s development.
Though there is no conclusive research that I’m aware of, it is probable that children establish certain growth limitations based on signals about nutrient availability received directly from their parents during conception and then from the mother during pregnancy and breastfeeding (variances of conveyed gut flora could be the mechanism here). Then, lifestyle and its epigenomic effects as normalized during childhood continues to play probably the same-seeming role since parents will tend to feed their children the same things they eat.
Anthropologically, going back a mere few hundred years there were no cultures anywhere in the world suffering obesity epidemics, so it doesn’t make sense to attribute variance too strongly to genetics. Historically, humans have survived healthfully on almost any combination of macronutrients while the main variant between healthy civilizations seems to have been micronutrients. Since studies generally don’t account in any fashion for idiosyncratic in-utero environment or for epigenetic variations among individuals, it could turn out that a vast amount of nutritional research is entirely worthless. E.g. clinical studies of nutrition among populations could depend entirely on sociological factors about the last generation’s diet than about the objective value of macro-nutrients (which, in my opinion, should never be claimed as the object of a study as if removed from the context of the foods they are a part of).
The father’s health can play a role after conception as well since beneficial gut bacteria, in the least, can be transferred through saliva & sex. Additionally, since these gut bacteria build up multi-generationally, it could be that antibiotic treatment seriously impairs the functioning of newborns, especially if they don’t have probiotic sources in their diet (the best of which is breastmilk from a biotics-rich mother!).
-med student
Which ought to be nearly obvious to anyone who has compared the calorie expenditures of common physical activities with the calorie contents of common foodstuffs. (Yes, increasing muscle mass increases thermogenesis (but so does caffeine) and I personally feel that doing abs help me feel less hungry because they kind of compress my stomach (but so does wearing higher-rise trousers and pulling their belt tighter), but those are second-order effects.)
This is also observed when wearing back-braces and corsets over the long term. In the corset-wearing/waist-training community particularly, some people have observed that without significant changes in behavior, corsets may decrease appetite; the actual effect is of course highly variable, but it’s frequent enough to be conventional wisdom in that community, so.