The chance of developing AIDS is probably also associated with age of HIV infection, the use of antiretroviral drugs—and other genetic and environmental factors.
Does the non-mutated CCR5 gene do anything important that the mutation disrupts? (In the sort of way the mutation for malaria resistance causes sickle cell anemia if you have two of them.)
That thesis shouldn’t be controversial either.
For instance, a person obtaining two mutated CCR5 genes will be virtually immune to the HIV virus.
The chance of developing AIDS is probably also associated with age of HIV infection, the use of antiretroviral drugs—and other genetic and environmental factors.
Does the non-mutated CCR5 gene do anything important that the mutation disrupts? (In the sort of way the mutation for malaria resistance causes sickle cell anemia if you have two of them.)