Using OxyContin(tm) for a job interview seems like a distinctly bad idea. Particularly if the employer asks for drug screening.. If you absolutely have to, I suggest sticking with Oxytocin.
I confused the substances and trusted outcomplete when I shouldn’t have. But what I said does apply to fascia speak about Oxytocin (my source is the book Anatomy Trains).
But not everyone does. Some doubt that nasal oxytocin even gets into the brain at all. Oxytocin is a peptide molecule, which means it can’t cross the blood-brain barrier and enter the central nervous system
The timeframe for the effect on fascia seems also to be about right (from Anatomy Trains):
The contraction, when it occurs, comes on very slowly
compared to any muscle contraction, building over 20-
30 minutes and sustaining for more than an hour before
slowly subsiding.
The neuroskeptic says:
many nasal oxytocin studies use a time delay of 30 or 45 minutes.
I think there is more evidence it crosses (two studies with spinal measures) than it does not (0 studies). For (almost) direct measures check out Neumann, Inga D., et al., 2013 and Born, 2002. There are great many studies showing effects that could only be caused by encephalic neuromodulation. If it does not cross it, then it should cause increased encephalic levels of some neurochemical with the exact same profile, but that would be really weird.
Using OxyContin(tm) for a job interview seems like a distinctly bad idea. Particularly if the employer asks for drug screening.. If you absolutely have to, I suggest sticking with Oxytocin.
I confused the substances and trusted outcomplete when I shouldn’t have. But what I said does apply to fascia speak about Oxytocin (my source is the book Anatomy Trains).
As far as the blood brain barrier goes: http://blogs.discovermagazine.com/neuroskeptic/2013/12/08/spinal-taps-save-oxytocin-research/#.VU-dufntlBc
The timeframe for the effect on fascia seems also to be about right (from Anatomy Trains):
The neuroskeptic says:
I think there is more evidence it crosses (two studies with spinal measures) than it does not (0 studies). For (almost) direct measures check out Neumann, Inga D., et al., 2013 and Born, 2002. There are great many studies showing effects that could only be caused by encephalic neuromodulation. If it does not cross it, then it should cause increased encephalic levels of some neurochemical with the exact same profile, but that would be really weird.