I think that the data required to figure out the gears of most major human age-related diseases is probably already available, online, today.
I find that wildly optimistic. My guess is quite opposite: while we see the symptoms of aging, and have some inkling of the underlying processes, like telomerase shortening, we have no clue at all about the underlying reasons for it, let alone about how this can be changed in a way that prolongs youth and delays the onset of aging.
I find that wildly optimistic. My guess is quite opposite: while we see the symptoms of aging, and have some inkling of the underlying processes, like telomerase shortening, we have no clue at all about the underlying reasons for it, let alone about how this can be changed in a way that prolongs youth and delays the onset of aging.
I don’t see how that contradicts his claim. Having the data required to figure out X is really not the same as knowing X.