This is actually not relevant as warfarin dosage is determined by regular testing and dose adjustment. Your inborn metabolic rate is a very small effect compared to, for example, dietary preferences. (for those who are unfamiliar with the agent, warfarin antagonises the effects of vitamin K and so must be adjusted against dietary intake)
Unfortunately there are many people in the health sector offering tests that, whilst factually correct, are irrelevant to a patient’s care.
The influence of the genotype varies from “typical sensitivity” to “may require greatly decreased warfarin dose”. A range that is all but irrelevant, regular testing or no (think for example of the initial dosage).
This is actually not relevant as warfarin dosage is determined by regular testing and dose adjustment. Your inborn metabolic rate is a very small effect compared to, for example, dietary preferences. (for those who are unfamiliar with the agent, warfarin antagonises the effects of vitamin K and so must be adjusted against dietary intake)
Unfortunately there are many people in the health sector offering tests that, whilst factually correct, are irrelevant to a patient’s care.
Someone tell the NHS, which is sponsoring a large trial to explore just that question.
The influence of the genotype varies from “typical sensitivity” to “may require greatly decreased warfarin dose”. A range that is all but irrelevant, regular testing or no (think for example of the initial dosage).