It doesn’t need to be anything complex. Perhaps just an neural activity statistics so that neurogenesis has information about where to put new neurons,
I generally think of this as a known unknown, I want more evidence that it is important before arguing too much. I think it just highlights that there might be unknown unknowns with regards to brain function, so our pdf for when ems occurs should be fairly broad.
Re-purposed unique binding ID proteins (forgot the biological term for it, each neuron makes itself entire series of more and less unique IDs).
Are you thinking about the immune system? If not I’d be interested in knowing more.
Yep, I think so too. Nothing in my reply to you right now may depend on the DNA directly as DNA is too slow; it has to use the already existing proteins which we already know we need to scan. Ditto for the ‘long term memory’; long term memory write and recall cannot rely on this if you remember something from 3 minutes ago (which is still ‘long term’).
So, from a scan that does not read DNA we would have sufficient information to at least create the brain emulation that can write this reply; we may also need to figure out how retrotransposons work in the neurons to make the simulation accurate long-term (the emulation might otherwise end up with some kind of learning disability, potentially quite severe if the emulated brain can’t retain long term memories beyond the span of several hours).
But I do not think we would absolutely have to find out a way to read the DNA off each neuron when making the scan. It would suffice to infer everything from the copy in the proteins.
(however if we find a way to read dna of every neuron, it might be that we could use it instead of reading the proteins themselves accurately).
Are you thinking about the immune system? If not I’d be interested in knowing more.
The system that makes neuron not connect to itself.
It doesn’t need to be anything complex. Perhaps just an neural activity statistics so that neurogenesis has information about where to put new neurons,
I generally think of this as a known unknown, I want more evidence that it is important before arguing too much. I think it just highlights that there might be unknown unknowns with regards to brain function, so our pdf for when ems occurs should be fairly broad.
Are you thinking about the immune system? If not I’d be interested in knowing more.
Yep, I think so too. Nothing in my reply to you right now may depend on the DNA directly as DNA is too slow; it has to use the already existing proteins which we already know we need to scan. Ditto for the ‘long term memory’; long term memory write and recall cannot rely on this if you remember something from 3 minutes ago (which is still ‘long term’).
So, from a scan that does not read DNA we would have sufficient information to at least create the brain emulation that can write this reply; we may also need to figure out how retrotransposons work in the neurons to make the simulation accurate long-term (the emulation might otherwise end up with some kind of learning disability, potentially quite severe if the emulated brain can’t retain long term memories beyond the span of several hours).
But I do not think we would absolutely have to find out a way to read the DNA off each neuron when making the scan. It would suffice to infer everything from the copy in the proteins. (however if we find a way to read dna of every neuron, it might be that we could use it instead of reading the proteins themselves accurately).
The system that makes neuron not connect to itself.
edit: that’s what i mean