Accessing long term memory appears to be a reconstructive process, which additionally results in accessed memories becoming fragile again; this is what I believe is occurring here. The learned aversion is reconstructed and as then susceptible to damage much more than other non-recently accessed LTM. Consider that the drug didn’t destroy ALL of the mice’s (fear?) memories, only that which was most recently accessed.
Accessing long term memory appears to be a reconstructive process, which additionally results in accessed memories becoming fragile again; this is what I believe is occurring here. The learned aversion is reconstructed and as then susceptible to damage much more than other non-recently accessed LTM. Consider that the drug didn’t destroy ALL of the mice’s (fear?) memories, only that which was most recently accessed.
So no worries to cryonics!