Specifically the folinic acid hypothesis is that this is an atypical autoimmune-instigated form of CFD, not the genetic type which typically manifests very rapidly. Look up Folate Receptor Alpha Autoantibodies. My strongest claim is that something environmental may be causing an uptick in autism, even accounting for its strong heritability; with a subset of that claim being “maybe vaccines”. I don’t believe this strongly enough to write more than a shortform pondering it.
The “rise” in autism’s prevalence is far more easily explain by a mix of better diagnosis and a more tolerant attitude towards autistic individuals allowing them to reproduce, especially when you consider that the rates of autism don’t differ all that much between the developed and the developing world
Plus the primary candidate mechanism for autisms symptoms is a lack of synaptic pruning not the lack of a particular nutrient
Specifically the folinic acid hypothesis is that this is an atypical autoimmune-instigated form of CFD, not the genetic type which typically manifests very rapidly. Look up Folate Receptor Alpha Autoantibodies. My strongest claim is that something environmental may be causing an uptick in autism, even accounting for its strong heritability; with a subset of that claim being “maybe vaccines”. I don’t believe this strongly enough to write more than a shortform pondering it.
I still don’t see this hypothesis going anywhere
The “rise” in autism’s prevalence is far more easily explain by a mix of better diagnosis and a more tolerant attitude towards autistic individuals allowing them to reproduce, especially when you consider that the rates of autism don’t differ all that much between the developed and the developing world
Plus the primary candidate mechanism for autisms symptoms is a lack of synaptic pruning not the lack of a particular nutrient